Preliminary experiments indicate conolidine could inhibit certain ion channels, lessening neuronal excitability and limiting ache indicators. This mechanism is particularly suitable in neuropathic suffering, where abnormal signaling causes persistent distress. Moreover, conolidine seems to affect G protein-coupled receptor (GPCR) pathways integral to pain perception. Computational doc... https://thomase036nkf6.scrappingwiki.com/user