Preliminary studies point out conolidine may inhibit precise ion channels, minimizing neuronal excitability and limiting agony signals. This mechanism is particularly relevant in neuropathic pain, wherever abnormal signaling causes persistent pain. Moreover, conolidine appears to influence G protein-coupled receptor (GPCR) pathways integral to soreness notion. Computational docking re... https://conradr480tmg6.livebloggs.com/profile